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1. 武汉大学中南医院基因诊断中心
2. 湖北医药学院生化教研室
纸质出版日期:2017,
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杨毅, 丁妍, 于爱清, 等. 自噬对宫颈癌细胞干性及增殖能力的影响[J]. 重庆医科大学学报, 2017,42(11):1390-1394.
Yang Yi, Ding Yan, Yu Aiqing, et al. Effects of autophagy on cervical cancer cells stemness and proliferative ability[J]. Journal of Chongqing Medical University, 2017,42(11):1390-1394.
杨毅, 丁妍, 于爱清, 等. 自噬对宫颈癌细胞干性及增殖能力的影响[J]. 重庆医科大学学报, 2017,42(11):1390-1394. DOI: 10.13406/j.cnki.cyxb.001130.
Yang Yi, Ding Yan, Yu Aiqing, et al. Effects of autophagy on cervical cancer cells stemness and proliferative ability[J]. Journal of Chongqing Medical University, 2017,42(11):1390-1394. DOI: 10.13406/j.cnki.cyxb.001130.
目的:研究自噬对宫颈癌细胞干性及增殖能力的影响。方法:采用悬浮培养法分离HeLa肿瘤干细胞
免疫荧光检测干细胞标志基因CD133和Nanog
吖啶橙染色观察自噬小体的形成;Western blot检测贴壁细胞和肿瘤干细胞中自噬相关基因Beclin1和LC3B的表达情况;贴壁细胞和肿瘤干细胞平衡盐溶液EBSS饥饿处理4 h和8 h后
吖啶橙染色观察自噬小体的形成
Western blot检测Beclin1和LC3B表达
实时细胞检测系统检测野生型和饥饿处理后HeLa细胞的增殖能力;自噬抑制剂3-methyladenine(3-MA)阻断宫颈癌贴壁细胞和干细胞的自噬后
Western blot检测Beclin1和LC3B表达
实时细胞检测系统检测野生型和自噬抑制剂处理后HeLa细胞的增殖能力;运用CRISPR/Cas9基因编辑技术降低自噬基因Beclin1的表达
通过实时细胞检测系统检测野生型和Beclin1突变HeLa细胞的增殖能力。结果:基础水平下
HeLa肿瘤干细胞比HeLa贴壁细胞自噬水平高。饥饿诱导处理后
HeLa肿瘤干细胞和HeLa贴壁细胞自噬水平均增高
均在4 h最强
8 h后有所减弱
且HeLa肿瘤干细胞在饥饿诱导各时间点均比HeLa贴壁细胞强;HeLa细胞饥饿处理4 h和8 h后增殖能力均有所下降
8 h下降更明显。应用3-MA后
HeLa肿瘤干细胞和HeLa贴壁细胞自噬均减弱
且HeLa贴壁细胞自噬减弱更为明显;细胞增殖能力也明显减弱。Beclin1突变后HeLa细胞较野生型HeLa细胞增殖能力增强。结论:自噬参与维持宫颈癌HeLa细胞的干性。同时
自噬也能影响宫颈癌HeLa细胞的增殖能力。通过基因打靶扰乱自噬关键基因(如Beclin1或LC3B等)而改变宫颈癌细胞自噬水平可为宫颈癌的治疗带来新的治疗策略。
Objective:To investigate the effect of autophagy on cervical cancer cells stemness and its proliferation ability. Methods:HeLa mammospheres were isolated with the method of suspension culture. Formation of autophagosome was observed by staining with acridine orange under fluorescence microscope. Western blot was used to detect the expression of Beclin1 and LC3 B in HeLa adherent cells and mammospheres. After starvation induction with EBSS in HeLa adherent cells and mammospheres for 4 hours and 8 hours
formation of autophagosome was observed by staining with acridine orange under fluorescence microscope;Western blot was used to detect the expression of Beclin1 and LC3 B
proliferative ability of wild type HeLa cells and starvation-stimulated HeLa cells was detected using a real-time cell-based assay system. After inhibiting the autophagy with 3-methyladenine(3-MA)in HeLa adherent cells and mammospheres
Western blot was used to detect the expression of Beclin1 and LC3 B and proliferative ability of wild type HeLa cells and 3-MA-stimulated HeLa cells was detected using a real-time cell-based assay system. CRISPR/Cas9 gene editing techniques was used to down regulate the expression of Beclin1
proliferative ability of wild type HeLa cells and Beclin1-mutated HeLa cells was detected using a real-time cell-based assay system. Results:Compared with HeLa adherent cells
HeLa mammospheres had higher basic levels of autophagy. And the autophagy level was higher in HeLa mammospheres than in adherent cells under the starvation conditions. The autophagy level is significantly higher after 4 hours starvation induction both in the mammospheres and the adherent cells
which was confirmed by western blot analysis and acridine orange fluorescent staining. The proliferative ability of starvation inducted HeLa cells were weakened compared with that of wild type HeLa cells and it’s more obvious at 8 hours. The expression of Beclin1 and LC3 B are lower in the presence of 3-MA and it’s more obvious in adherent cells. The proliferative ability of HeLa cells were also weakened in the presence of 3-MA. Additionally
the proliferative ability of Beclin1-mutated HeLa cells were elevated compared with that of wild type HeLa cells. Conclusion:Autophagy is involved in maintaining the stemness of cervical cancer cells. Significantly
autophagy also can have an impact on the proliferative ability of cervical cancer cells. Gene targeting-induced disruption of autophagy key gene Beclin1 and LC3 B to alter autophagy can be a promising therapeutic strategy for curing cervical cancer.
自噬宫颈癌干细胞Beclin1LC3B
autophagycervical cancer cellsBeclin1LC3B
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