Interruption of bile acid enterohepatic circulation inhibits glycogen synthesis and promotes hepatocellular carcinoma progression

Jiyan Wang; Xintong Dai; Hongkai Chang; Qingle Gao; Jianshuang Guo; Juze Yang; Shuai Zhang; Changliang Shan

doi:10.1016/j.gendis.2023.02.026

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Interruption of bile acid enterohepatic circulation inhibits glycogen synthesis and promotes hepatocellular carcinoma progression

Rapid Communications | 更新时间：2024-02-04

- Interruption of bile acid enterohepatic circulation inhibits glycogen synthesis and promotes hepatocellular carcinoma progression
- Interruption of bile acid enterohepatic circulation inhibits glycogen synthesis and promotes hepatocellular carcinoma progression
- Genes & Diseases 2023年1卷第6期页码：2218-2221
- 作者机构：
  
  1. State Key Laboratory of Medicinal Chemical Biology, College of Pharmacy and Tianjin Key Laboratory of Molecular Drug Research, Nankai University,Tianjin,China,300350
  2. Department of Respiratory Medicine, Sir Run Run Shaw Hospital and Institute of Translational Medicine, Zhejiang University School of Medicine,Zhejiang,Hangzhou,China,310016
  3. School of Integrative Medicine, Tianjin University of Traditional Chinese Medicine,Tianjin,China,301617
- 作者简介：
- 基金信息：
- DOI：10.1016/j.gendis.2023.02.026
  中图分类号：
- 纸质出版日期：2023，
  
  网络出版日期：2023-11-1，
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Interruption of bile acid enterohepatic circulation inhibits glycogen synthesis and promotes hepatocellular carcinoma progression[J]. Genes & Diseases, 2023,1(6):2218-2221.

Jiyan Wang, Xintong Dai, Hongkai Chang, et al. Interruption of bile acid enterohepatic circulation inhibits glycogen synthesis and promotes hepatocellular carcinoma progression[J]. Genes & Diseases, 2023,1(6):2218-2221.
Interruption of bile acid enterohepatic circulation inhibits glycogen synthesis and promotes hepatocellular carcinoma progression[J]. Genes & Diseases, 2023,1(6):2218-2221. DOI： 10.1016/j.gendis.2023.02.026.

Jiyan Wang, Xintong Dai, Hongkai Chang, et al. Interruption of bile acid enterohepatic circulation inhibits glycogen synthesis and promotes hepatocellular carcinoma progression[J]. Genes & Diseases, 2023,1(6):2218-2221. DOI： 10.1016/j.gendis.2023.02.026.

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1 S.L. Chen, C.Z. Zhang, L.L. Liu, et al.A GYS2/p53 negative feedback loop restricts tumor growth in HBV-related hepatocellular carcinoma Cancer Res, 79 (3) (2019), pp. 534-545

2 L. Chen, T. Jiao, W. Liu, et al.Hepatic cytochrome P450 8B1 and cholic acid potentiate intestinal epithelial injury in colitis by suppressing intestinal stem cell renewal Cell Stem Cell, 29 (9) (2022), pp. 1366-1381

3 K. Husain, R.A. AnsariInfluence of cholinergic and adrenergic blocking drugs on hyperglycemia and brain glycogenolysis in diazinon-treated animals Can J Physiol Pharmacol, 66 (9) (1988), pp. 1144-1147

4 H. Aramjoo, T. Farkhondeh, M. Aschner, et al.The association between diazinon exposure and dyslipidemia occurrence: a systematic and meta-analysis study Environ Sci Pollut Res, 28 (4) (2021), pp. 3994-4006

5 Y. Zhang, F. Chen, D.S. Chandrashekar, et al.Proteogenomic characterization of 2002 human cancers reveals pan-cancer molecular subtypes and associated pathways Nat Commun, 13 (1) (2022), p. 2669

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