Short-term nitisinone discontinuation of hereditary tyrosinemia type 1 mice causes metabolic alterations in glutathione metabolism/biosynthesis and multiple amino acid degradation pathways

Colemonts-Vroninks Haaike; Norman P. Brendan; Van Laere Sven; Davison S. Andrew; Marcélis Lionel; Casimir Georges; Goyens Philippe; Claes Paul; De Bundel Dimitri; Martens Geert; Ranganath Lakshminarayan Rao; Vanhaecke Tamara; Gallagher James A; De Kock Joery

doi:10.1016/j.gendis.2022.11.013

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Short-term nitisinone discontinuation of hereditary tyrosinemia type 1 mice causes metabolic alterations in glutathione metabolism/biosynthesis and multiple amino acid degradation pathways

Short communication | 更新时间：2023-08-29

- Short-term nitisinone discontinuation of hereditary tyrosinemia type 1 mice causes metabolic alterations in glutathione metabolism/biosynthesis and multiple amino acid degradation pathways
- Short-term nitisinone discontinuation of hereditary tyrosinemia type 1 mice causes metabolic alterations in glutathione metabolism/biosynthesis and multiple amino acid degradation pathways
- Genes & Diseases 2023年10卷第5期
- 作者机构：
  
  1. Liverpool University Hospitals NHS Foundation Trust, Clinical Biochemistry and Metabolic Medicine, Liverpool L3 5PS, United Kingdom
  2. Ghent University, Department of Biomolecular Medicine, Ghent,Belgium,9052
  3. Vrije Universiteit Brussel, In Vitro Liver Disease Modelling Team, In Vitro Toxicology and Dermato-Cosmetology (IVTD) Research Group, Faculty of Medicine and Pharmacy, Laarbeeklaan 103,B,Brussels,Belgium,1090
  4. University of Liverpool, Dept. of Musculoskeletal & Ageing Science, Institute of Life Course & Medical Sciences, Liverpool L7 8TX, United Kingdom
  5. Vrije Universiteit Brussel, Liver Therapy & Evolution Team, In Vitro Toxicology and Dermato-Cosmetology (IVTD) Research Group, Faculty of Medicine and Pharmacy, Laarbeeklaan 103,B,Brussels,Belgium,1090
  6. Vrije Universiteit Brussel, Interfaculty Center Data Processing and Statistics, Faculty of Medicine and Pharmacy, Laarbeeklaan 103,B,Brussels,Belgium,1090
  7. Université Libre de Bruxelles, Laboratoire de Pédiatrie,B,Brussels,Belgium,1050
  8. Vrije Universiteit Brussel, Experimental Pharmacology (EFAR) Research Group, Faculty of Medicine and Pharmacy, Laarbeeklaan 103,B,Brussels,Belgium,1090
  9. AZ Delta General Hospital, Department of Laboratory Medicine, Roeselare,Belgium,8800
- 作者简介：
- 基金信息：
- DOI：10.1016/j.gendis.2022.11.013
  中图分类号：
- 纸质出版日期：2023，
  
  网络出版日期：2023-9-1，
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Colemonts-Vroninks Haaike, Norman P. Brendan, Van Laere Sven, 等. Short-term nitisinone discontinuation of hereditary tyrosinemia type 1 mice causes metabolic alterations in glutathione metabolism/biosynthesis and multiple amino acid degradation pathways[J]. Genes & Diseases, 2023,10(5).

Colemonts-Vroninks Haaike, Norman P. Brendan, Van Laere Sven, et al. Short-term nitisinone discontinuation of hereditary tyrosinemia type 1 mice causes metabolic alterations in glutathione metabolism/biosynthesis and multiple amino acid degradation pathways[J]. Genes & Diseases, 2023,10(5).
Colemonts-Vroninks Haaike, Norman P. Brendan, Van Laere Sven, 等. Short-term nitisinone discontinuation of hereditary tyrosinemia type 1 mice causes metabolic alterations in glutathione metabolism/biosynthesis and multiple amino acid degradation pathways[J]. Genes & Diseases, 2023,10(5). DOI： 10.1016/j.gendis.2022.11.013.

Colemonts-Vroninks Haaike, Norman P. Brendan, Van Laere Sven, et al. Short-term nitisinone discontinuation of hereditary tyrosinemia type 1 mice causes metabolic alterations in glutathione metabolism/biosynthesis and multiple amino acid degradation pathways[J]. Genes & Diseases, 2023,10(5). DOI： 10.1016/j.gendis.2022.11.013.

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Short-term nitisinone discontinuation of hereditary tyrosinemia type 1 mice causes metabolic alterations in glutathione metabolism/biosynthesis and multiple amino acid degradation pathways

Short-term nitisinone discontinuation of hereditary tyrosinemia type 1 mice causes metabolic alterations in glutathione metabolism/biosynthesis and multiple amino acid degradation pathways

DOI：10.1016/j.gendis.2022.11.013

摘要

Abstract

关键词

Keywords

references