Haploinsufficiency of KCNAB3 causes sudden cardiac death through delayed inactivation of Kv1.5 channel upon depolarization and induced myocardial apoptosis
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Short communication|更新时间:2023-08-29
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Haploinsufficiency of KCNAB3 causes sudden cardiac death through delayed inactivation of Kv1.5 channel upon depolarization and induced myocardial apoptosis
Haploinsufficiency of KCNAB3 causes sudden cardiac death through delayed inactivation of Kv1.5 channel upon depolarization and induced myocardial apoptosis
Genes & Diseases2023年10卷第3期
作者机构:
1. Department of Anesthesiology, Peking Union Medical College Hospital, Chinese Academy of Medical Sciences,Beijing,China,100005
2. Department of Respiratory Medicine, Diagnosis and Treatment Center of Respiratory Disease, The Second Xiangya Hospital of Central South University,Hunan,Changsha,China,410011
3. Department of Anesthesiology, The Second Xiangya Hospital of Central South University,Hunan,Changsha,China,410011
He-Yu Ji, Zhao-Jing Lin, Chan Chen, 等. Haploinsufficiency of KCNAB3 causes sudden cardiac death through delayed inactivation of Kv1.5 channel upon depolarization and induced myocardial apoptosis[J]. Genes & Diseases, 2023,10(3).
He-Yu Ji, Zhao-Jing Lin, Chan Chen, et al. Haploinsufficiency of KCNAB3 causes sudden cardiac death through delayed inactivation of Kv1.5 channel upon depolarization and induced myocardial apoptosis[J]. Genes & Diseases, 2023,10(3).
He-Yu Ji, Zhao-Jing Lin, Chan Chen, 等. Haploinsufficiency of KCNAB3 causes sudden cardiac death through delayed inactivation of Kv1.5 channel upon depolarization and induced myocardial apoptosis[J]. Genes & Diseases, 2023,10(3). DOI: 10.1016/j.gendis.2022.05.023.
He-Yu Ji, Zhao-Jing Lin, Chan Chen, et al. Haploinsufficiency of KCNAB3 causes sudden cardiac death through delayed inactivation of Kv1.5 channel upon depolarization and induced myocardial apoptosis[J]. Genes & Diseases, 2023,10(3). DOI: 10.1016/j.gendis.2022.05.023.
Haploinsufficiency of KCNAB3 causes sudden cardiac death through delayed inactivation of Kv1.5 channel upon depolarization and induced myocardial apoptosis